Methamphetamine neurotoxicity, microglia, and neuroinflammation

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Role of microglia in methamphetamine-induced neurotoxicity.

Methamphetamine (Meth) is an addictive psychostimulant widely abused around the world. The chronic use of Meth produces neurotoxicity featured by dopaminergic terminal damage and microgliosis, resulting in serious neurological and behavioral consequences. Ample evidence indicate that Meth causes microglial activation and resultant secretion of pro-inflammatory molecules leading to neural injury...

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Diesel Exhaust Activates and Primes Microglia: Air Pollution, Neuroinflammation, and Regulation of Dopaminergic Neurotoxicity

BACKGROUND Air pollution is linked to central nervous system disease, but the mechanisms responsible are poorly understood. OBJECTIVES Here, we sought to address the brain-region-specific effects of diesel exhaust (DE) and key cellular mechanisms underlying DE-induced microglia activation, neuroinflammation, and dopaminergic (DA) neurotoxicity. METHODS Rats were exposed to DE (2.0, 0.5, and...

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Methamphetamine (METH) is a potent psychostimulant with neurotoxic properties. Heavy use increases the activation of neuronal nitric oxide synthase (nNOS), production of peroxynitrites, microglia stimulation, and induces hyperthermia and anorectic effects. Most METH recreational users also consume cannabis. Preclinical studies have shown that natural (Δ9-tetrahydrocannabinol, Δ9-THC) and synthe...

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ژورنال

عنوان ژورنال: Journal of Neuroinflammation

سال: 2018

ISSN: 1742-2094

DOI: 10.1186/s12974-018-1385-0